RESEARCH

Tubulovesicle trafficking and exocytosis during gastric acid secretion by parietal cells.

Enteroendocrine hormone cholecystokinin-induced Ca²⁺ increase in intracellular calcium in enteric glial cells (Cells were loaded with Fluo 4-AM Ca²⁺ sensing dyes).

Forskolin-induced Ca²⁺ release from tubulovesicles in parietal cells (Genetically encoded Ca²⁺sensor GCaMP3 was expressed in mouse parietal cells).

Removal of fast N-type inactivation of A-type K⁺ channels by pharmacological modulators.

The current-voltage relationship (I−V curve) of voltage-gated K⁺ channels.

Pharmacological inhibition of voltage-gated K⁺ channels.

Structure and function of the A-type K⁺ channels ball domain (amino acids 1-62).